In the case of multiple sclerosis, the human immune system acts in such a way that its efforts destroy myelin, which is an insulated layer at the end of the neurons of the spinal cord and brain, as well as the spinal cord. As a result, these neurons – and, consequently, the related departments of these areas – stop working correctly, which worsens the patient's condition. However, a team of specialists from the University of Oregon in the United States today presented its new study on the study of the so-called molecule of the chemist, invented about twenty years ago to lower cholesterol levels – however, it can cope with sclerosis.
These conclusions were reached by experts shortly after they conducted preliminary tests on experimental mice, which had previously been genetically induced, symptoms associated with multiple sclerosis. And then, by introducing an experimental molecule of the obstetroma into their brain neurons, specialists observed the subsequent process of improving their condition. And so it happened – this connection was really actively and effectively engaged in the restoration of the myelin sheath of neurons.
It should, however, be clarified that the scientists had previously somewhat improved the effect of the molecule, giving it the ability to effectively bypass the traditional brain and blood barrier, complementing it with a less experimental Sob-AM2 molecule, through which the compound could break through the natural brain flaps. As a result, the degree of recovery of myelin reached really high rates, which could not fail to please the development specialists from the University of Oregon.
In addition, in the future, experts plan to further improve the efficiency and effectiveness of this molecule – it is known that the prototype of the working draft and all its preliminary results have already been transferred to the research company Llama Theraupetics, which gives hope for an accelerated development of new iterations of such anti-sclerosis drugs basis of an experimental experimental molecule.